Study sheds light on link between cannabis, anxiety and stress
Jan. 15, 2020—A molecule produced by the brain that activates the same receptors as marijuana is protective against stress by reducing anxiety-causing connections between two brain regions.
Personalized pain management
Feb. 28, 2019—Understanding how natural brain chemicals with pain-relieving properties interact with administered opioids may guide personalized approaches for pain management.
Cannabis compound reduces seizures
Feb. 26, 2018—Cannabidiol (CBD) oils reduced seizures in patients with difficult-to-treat epilepsy, Vanderbilt investigators have found.
Regulating anxiety in the brain
Apr. 28, 2017—Two brain signaling pathways have overlapping functions in regulating anxiety, suggesting that therapeutics aimed at one or the other will impact both.
Vanderbilt study finds natural chemical helps brain adapt to stress
Mar. 28, 2017—A natural signaling molecule that activates cannabinoid receptors in the brain plays a critical role in stress-resilience — the ability to adapt to repeated and acute exposures to traumatic stress, according to researchers at Vanderbilt University Medical Center.
Natural ‘high’ could avoid chronic marijuana use, Vanderbilt study finds
Dec. 1, 2014—Replenishing the supply of a molecule that normally activates cannabinoid receptors in the brain could relieve mood and anxiety disorders and enable some people to quit using marijuana.
Reversing stress-induced anxiety
Jul. 21, 2014—Augmenting the signals of natural “endocannabinoids” in the brain may be a promising approach for treating mood and anxiety disorders.
Discovery sheds new light on marijuana’s anxiety relief effects
Mar. 6, 2014—An international group led by Vanderbilt University researchers has found cannabinoid receptors, through which marijuana exerts its effects, in a key emotional hub in the brain involved in regulating anxiety and the flight-or-fight response.
Toxin provides endometriosis clues
Nov. 2, 2012—Environmental toxins may contribute to endometriosis-related infertility by disrupting endometrial response to progesterone.