journal publication Archives
‘Longevity’ gene has role in bone loss
Dec. 6, 2012—A gene associated with long life participates in the normal regulation of bone remodeling and may have a role in bone loss associated with aging.
Carcinogenic chemicals cramp DNA
Nov. 21, 2012—Researchers have determined how a DNA lesion caused by exposure to chemicals may spark cancer formation.
Bird’s eye view of gut development
Nov. 19, 2012—A new map of the developing gut of quail offers valuable tool for studying intestinal diseases.
Green tea for failing hearts?
Nov. 16, 2012—The main antioxidant compound in green tea increases contractile force in isolated heart cells, suggesting it may be useful in heart failure.
Inherited lung disease no worse in offspring
Nov. 15, 2012—An inherited lung disease does not appear to have earlier onset and increased severity – a phenomenon called genetic anticipation – in successive generations.
Kidney woes during heart failure
Nov. 8, 2012—Levels of the protein NGAL may be a good predictor of worsening kidney function in patients with acute heart failure.
Bacterial signals in sarcoidosis
Nov. 6, 2012—Study adds evidence for infectious agents’ role in sarcoidosis, an inflammatory condition that can lead to respiratory failure and death.
Toxin provides endometriosis clues
Nov. 2, 2012—Environmental toxins may contribute to endometriosis-related infertility by disrupting endometrial response to progesterone.
Protein’s length, flexibility key to infection
Oct. 31, 2012—The structural architecture of the reovirus attachment protein is required for efficient reovirus infection of host cells.
Bid protein guards blood cells against stress
Oct. 25, 2012—A protein normally known to promote cell death found to encourage survival of blood forming cells.
Stomach bug alters tumor suppressor
Oct. 23, 2012—The stomach bug Helicobacter pylori increases forms of a protein that promote tumor development, perhaps explaining how it elevates risk for gastric cancer.
Setting traps to probe gene function
Oct. 18, 2012—A new method for creating genetic mutations that can be activated at certain times or in specific tissues will enable studies to probe gene function.
