Neurofibromatosis Archives
Study seeks to devise tools to evaluate changes in bone quality
Jul. 12, 2018—Sometimes it makes sense to put the cart in front of the horse.
VICC to host symposium on neurofibromatosis April 21
Apr. 12, 2018—Vanderbilt-Ingram Cancer Center (VICC) will host a special event for patients and families affected by neurofibromatosis (NF), a genetic disorder that can cause tumors in the nervous system. The Neurofibromatosis Patient and Family Symposium will be held Saturday, April 21, 9 a.m. – 1 p.m., at the VICC 8th floor conference center in the Preston Research Building, 2220 Pierce Ave.
VICC to host neurofibromatosis event April 21
Apr. 5, 2018—Vanderbilt-Ingram Cancer Center (VICC) will host a special event for patients and families affected by neurofibromatosis (NF), a genetic disorder that can cause tumors in the nervous system.
Research participants needed for neurofibromatosis study focused on reading difficulties
Mar. 1, 2018—The Vanderbilt Education and Brain Sciences Research Lab is seeking participants age 8 to 20 for a research study focusing on reading difficulties and neurofibromatosis.
Vanderbilt joins national neurofibromatosis network
Nov. 9, 2017—The Vanderbilt-Ingram Cancer Center Neurofibromatosis (NF) Clinic has joined the Children’s Tumor Foundation NF Clinic Network.
Neurofibromin fine-tunes bone growth
May. 6, 2015—The protein neurofibromin acts as a brake in a signaling pathway that is important in bone development, Vanderbilt researchers have discovered.
Bone healing therapy for NF1 fractures
Sep. 11, 2014—A combination treatment delivered to the site of fractures may improve bone healing in patients with the genetic disease neurofibromatosis type-1.
Novel treatment strengthens bones in genetic disease
Aug. 7, 2014—An enzyme therapy may prevent skeletal abnormalities associated with the genetic disorder neurofibromatosis type-1, Vanderbilt investigators have discovered.
Factor’s role in long bone development
Jul. 31, 2013—Insight into how the protein neurofibromin participates in the signaling pathway that produces the body’s long bones has implications for fracture healing in some patients.
Skeletal defects in genetic disorder
Nov. 18, 2011—A new mouse model provides a tool for testing novel therapeutic approaches for neurofibromatosis.