Immune cells that produce an anti-inflammatory factor are enriched in fat tissue around the heart and may be good targets to improve heart attack outcomes.

Loss of a protein that regulates mitochondrial function can greatly increase the risk of myocardial infarction (heart attack), Vanderbilt scientists reported Oct. 3 in the journal eLife.

A Vanderbilt-led research team has discovered genetic variations that increase the risk of heart attack even when patients are receiving a statin drug like Lipitor or Crestor to lower their blood cholesterol.

A national effort seeks to raise awareness about an uncommon cause of heart attacks in otherwise healthy, young and middle-aged women.

Vanderbilt University Medical Center investigators have identified a growing number of serious and sometimes fatal cases of heart problems among cancer patients treated with some forms of immunotherapy.

When Jessica Biggs first met Esther Kim, M.D., associate professor of Medicine at Vanderbilt, the 33-year-old burst into tears.

A protein called Gremlin 2 controls the extent of inflammation after heart attack and may be a good therapeutic target.