Timothy Hohman Archive — Page 1 of 1

Blue side profile of a human head silhouette containing interlocking colorful gears, symbolizing brain function and the workings of the human body.
November 8, 2022

Alzheimer’s risk factor and cognition

Vanderbilt researchers found that a protein with roles in innate immunity worsens memory at baseline in carriers of APOE-e4, the strongest genetic risk factor for Alzheimer’s disease, further implicating neuroinflammation in cognitive decline.

Blue side-profile silhouette of a human head and neck filled with interlocking gears, with additional gears floating above and to the right.
October 1, 2021

VUMC awarded $31.7 million to harmonize Alzheimer’s research data

Vanderbilt University Medical Center has received a $31.7 million federal grant to harmonize research data gathered on human subjects in scores of disparate studies of Alzheimer’s disease and related dementias.

Older woman touching the area around her eye while another person rests a hand on her shoulder.
September 3, 2020

New clue to Alzheimer’s disease

Combining studies of genetically diverse mouse populations and human data led to the identification of a gene associated with cognitive decline and brain changes in Alzheimer’s disease.

May 7, 2018

Study provides robust evidence of sex differences with Alzheimer’s gene

The APOE gene, the strongest genetic risk factor for Alzheimer’s disease, may play a more prominent role in disease development among women than men, according to new research from the Vanderbilt Memory and Alzheimer’s Center.

November 10, 2016

Alzheimer’s study establishes way to measure resilience

Vanderbilt researchers have established a new measure of resilience to cognitive impairment in people with asymptomatic Alzheimer’s disease.

Overlapping word cards feature “Alzheimers” prominently, surrounded by terms including “Delusion,” “Memory Loss,” “Dementia,” “Depression,” and “Irritability.”
April 10, 2014

Study explores genetics behind Alzheimer’s resiliency

Autopsies have revealed that some individuals develop the cellular changes indicative of Alzheimer’s disease without ever showing clinical symptoms in their lifetime.