Understanding how a bacterial protein that influences the risk of stomach cancer is produced could guide new strategies for treatment.

The bacterium H. pylori is a leading cause of stomach cancer, and Vanderbilt researchers are studying how one of its toxins gets into cells.

The bacterium Helicobacter pylori has been linked to anemia and may be contributing to the incidence and severity of anemia worldwide.

Vanderbilt researchers show that a certain factor negatively impacts the first-line responder cells in the lungs, providing one explanation for why patients with asthma are at greater risk for invasive bacterial disease.

Understanding factors, such as the receptor TLR9, that detect and defend against pathogens may lead to therapeutic approaches that promote an effective immune response to treat infections.

The latest research on pneumonic plague, respiratory syncytial virus (RSV) and other life-threatening infections will be discussed during a Symposium on Infection and Immunity April 11 at the Vanderbilt Student Life Center.

A high-salt diet worsens the carcinogenic effects of Helicobacter pylori, a bacterium that colonizes the stomachs of half of the world’s population.