A person’s mitochondrial gene “signature” could predict risk for diabetic retinopathy and guide early intervention strategies.

Understanding the cellular and molecular responses of the eye to blast injury could guide new treatment development.

An ion channel protein called TRIPV1 helps retinal neurons survive the elevated eye pressure associated with glaucoma.

MRI brain scans reveal that children with autism spectrum disorders respond to a narrower range of familiar rewards.

Vanderbilt researchers are studying how mutations in the receptor for light, rhodopsin, cause light blindness.

A panel of metabolites – small molecules that are part of metabolic processes – that are unique to macular degeneration will shed light on the disease and aid diagnosis.

Glaucoma-causing mutations in the gene for myocilin reduce secretion of the protein into the aqueous humor, suggesting a new option for treatment.