Jun. 29, 2018—Lipid molecules that bind to HDL can modify its function — and blocking that modification can protect HDL and potentially lower the risk of atherosclerosis and heart disease.

An enzyme in the gut – NAPE-PLD – is required for the action of signaling molecules that reduce food intake and weight gain.

Sean Davies, Ph.D., associate professor of Pharmacology, was recognized as Pharmacology “Teacher of the Year” during the 25th Annual Joel G. Hardman Student-Invited Pharmacology Forum in Light Hall.

In some cases, HDL, the "good cholesterol," may not function properly and may actually accelerate atherosclerosis.

Engineered bacteria that produce a therapeutic compound in the gut prevent obesity in mice, Vanderbilt University investigators have discovered.