Oxidative stress and toxic products called isolevuglandins in mitochondria play a role in endothelial dysfunction and hypertension — and getting rid of them with a special “scavenger” molecule has therapeutic potential.

Excess dietary salt activates immune cells to induce inflammation and hypertension, supporting current recommendations for low sodium consumption.

A new Clinical Fellowship in Autonomic Disorders, administered through the Division of Clinical Pharmacology in the Department of Medicine at Vanderbilt University Medical Center, will be one of only four accredited fellowship programs in autonomic disorders in the country.

Vanderbilt University Medical Center investigators have used high-throughput robotic technology to rapidly study and classify variations in a gene linked to heart rhythm disorders and cardiac conditions.

Vanderbilt researchers have discovered that a protein called SGK1 in immune cells is activated by sodium, leading to the development of salt-sensitive hypertension.

John Oates, MD, an internationally known physician at Vanderbilt University School of Medicine and a founding father of the discipline of Clinical Pharmacology, died Tuesday (July 30) in Nashville after a short illness. He was 87.

Meena Madhur, MD, PhD, has received a Presidential Early Career Award for Scientists and Engineers.