Research Archive — Page 119 of 131

Insulin in vials
December 8, 2016

Making human beta cells reproduce

A new method developed at Vanderbilt will speed the search for potential therapeutics for diabetes: compounds that stimulate the replication of insulin-producing beta cells.

By VUMC News and Communications
December 1, 2016

Investigators explore new way to control mosquitoes

In a new study, Vanderbilt pharmacologist Jerod Denton, Ph.D., Ohio State entomologist Peter Piermarini, Ph.D., and colleagues report an experimental molecule that inhibits kidney function in mosquitoes and thus might provide a new way to control the deadliest animal on Earth.

By Paul Govern
November 28, 2016

Rockefeller University’s Elaine Fuchs, Ph.D., named 2016 recipient of the Vanderbilt Prize in Biomedical Science

Elaine Fuchs, Ph.D., whose innovative use of reverse genetics has helped redefine the study of skin diseases and cancer stem cells, is the recipient of the 2016 Vanderbilt Prize in Biomedical Science, officials at Vanderbilt University Medical Center (VUMC) announced today.

By Bill Snyder
brain and lightning
November 10, 2016

Protein structure and epilepsy severity

Understanding how mutations affect the structure and function of inhibitory neurotransmitter receptors will shed light on the mechanisms underlying some types of epilepsy.

By VUMC News and Communications
November 7, 2016

Early study finds antibody that ‘neutralizes’ Zika virus

Researchers at Vanderbilt University Medical Center and Washington University School of Medicine in St. Louis have isolated a human monoclonal antibody that in a mouse model “markedly reduced” infection by the Zika virus.

By Bill Snyder
tug-of-rope pair
November 3, 2016

VUMC investigators find pathogens work together to infect host

Pseudomonas aeruginosa and Staphylococcus aureus — two pathogens that frequently co-infect the lungs of patients with cystic fibrosis — appear to cooperate with each other, Vanderbilt investigators have discovered. When pseudomonas is starved for metal by the host, it shuts down the production of factors that would normally kill staph, promoting a co-infection.

By Leigh MacMillan