Research Archive — Page 120 of 133
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January 12, 2017
Nominations sought for Vanderbilt Prize in Biomedical Science
Nominations for the 2017 Vanderbilt Prize in Biomedical Science are now being accepted. The deadline is March 31. -
December 8, 2016
Making human beta cells reproduce
A new method developed at Vanderbilt will speed the search for potential therapeutics for diabetes: compounds that stimulate the replication of insulin-producing beta cells. -
December 1, 2016
Investigators explore new way to control mosquitoes
In a new study, Vanderbilt pharmacologist Jerod Denton, Ph.D., Ohio State entomologist Peter Piermarini, Ph.D., and colleagues report an experimental molecule that inhibits kidney function in mosquitoes and thus might provide a new way to control the deadliest animal on Earth. -
November 28, 2016
Rockefeller University’s Elaine Fuchs, Ph.D., named 2016 recipient of the Vanderbilt Prize in Biomedical Science
Elaine Fuchs, Ph.D., whose innovative use of reverse genetics has helped redefine the study of skin diseases and cancer stem cells, is the recipient of the 2016 Vanderbilt Prize in Biomedical Science, officials at Vanderbilt University Medical Center (VUMC) announced today. -
November 10, 2016
Protein structure and epilepsy severity
Understanding how mutations affect the structure and function of inhibitory neurotransmitter receptors will shed light on the mechanisms underlying some types of epilepsy. -
November 7, 2016
Early study finds antibody that ‘neutralizes’ Zika virus
Researchers at Vanderbilt University Medical Center and Washington University School of Medicine in St. Louis have isolated a human monoclonal antibody that in a mouse model “markedly reduced” infection by the Zika virus. -
November 3, 2016
VUMC investigators find pathogens work together to infect host
Pseudomonas aeruginosa and Staphylococcus aureus — two pathogens that frequently co-infect the lungs of patients with cystic fibrosis — appear to cooperate with each other, Vanderbilt investigators have discovered. When pseudomonas is starved for metal by the host, it shuts down the production of factors that would normally kill staph, promoting a co-infection.